After reading this chapter, you will be able to: • List the clinical manifestations common with newborn and early childhood respiratory disorders, including the following: • Clinical manifestations associated with increased negative intrapleural pressures during inspiration • Flaring nostrils (or nasal flaring) • Describe the meaning of apnea of prematurity. • List factors that trigger apnea in the premature infant. • Describe persistent pulmonary hypertension of the newborn (PPHN). • Describe the arterial blood gas values commonly associated with newborn respiratory disorders, and include the three major mechanisms responsible for the decreased Pao2 associated with newborn pulmonary disorders. • Discuss the objective data, assessments, and treatment plans commonly associated with newborn respiratory disorders. • Describe the major components of the Apgar score. • Define key terms and complete self-assessment questions at the end of the chapter and on Evolve. The thorax of the newborn infant is quite flexible—that is, the compliance of the infant’s thorax is high. This flexibility is a result of the large amount of cartilage found in the skeletal structure of newborns. Because of the structural alterations associated with many newborn respiratory disorders, however, the compliance of the infant’s lungs is low. In an effort to offset the decreased lung compliance, the infant must generate more negative intrapleural pressures during inspiration. This condition causes the following (see Figure 31-1): • The soft tissues between the ribs retract during inspiration. • The substernal area retracts and the abdominal area protrudes in a seesaw fashion during inspiration. The substernal retraction is caused by high negative intrapleural pressure, and the abdominal distention is caused by the contraction (depression) of the diaphragm during inspiration. • The blood vessels in the more dependent portions of the thoracic and abdominal areas dilate and pool blood, causing these areas to appear cyanotic. Flaring nostrils (or nasal flaring) frequently are observed in infants in respiratory distress. This clinical manifestation probably is a facial reflex to facilitate the movement of gas into the tracheobronchial tree. The dilator naris, which originates from the maxilla and inserts into the ala of the nose, is the muscle responsible for this movement. When activated, the dilator naris pulls the alae laterally and widens the nasal aperture, providing a larger orifice for gas to enter during inspiration (Figure 31-2).
Newborn and Early Childhood Respiratory Disorders
Clinical Manifestations Common with Newborn and Early Childhood Respiratory Disorders
Clinical Manifestations Associated with More Negative Intrapleural Pressures during Inspiration
Flaring Nostrils (or Nasal Flaring)
Newborn and Early Childhood Respiratory Disorders
