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Secondary hyperaldosteronism
- 2. DEFINITION Common clinical condition occurs secondary to some cause which stimulate RAAS. It is either physiological or pathological
- 3. ETIOLOGY Physiologic causes – Salt depletion- inadequate salt intake, excessive salt through kidney or GI Pregnancy- progesterone stimulate aldosterone production pathologic causes- Hypoperfusion of kidney – nephrotic syndrome, liver cirrhosis, CHF, diuretic therapy
- 4. Renal ischemia- malignant hypertension,RAS High renin production by juxta glomerular apparatus
- 5. DIAGNOSIS Plasma sodium level- normal or near normal Plasma potassium level – low Plasma renin and aldosterone- elevated
- 6. MANAGEMENT TREAT UNDERLYING CAUSE Salt depletion- normal saline infusion Nephrotic syndrome, cirrohsis of liver CHF, -SPIRONOLACTONE+ LOOP DIURETIC Renal ischemia- ACE INHIBITORS RAS- TRANSLUMINAL ANGIOPLASTY Diuretic therapy- use potassium sparing diuretics.
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