2. Definition:
An increased pressure within enclosed
osteofascial space that reduces capillary per-
fusion below level necessary for tissue
viability; the underlying mechanism is:
- increased volume within space
- decreased space for contents
- combination of both
3. History
1881-Volkman described contracted state believed due to
ischemic muscle
1884-Lesser developed clinical model
1888-Peterson felt due to nerve compromise
1906-Hildebrand coined “Volkman’s ischemic contracture”
1914-Murphy recommended fasciotomy to prevent contracture
1940-Griffiths ‘4 Ps’
1966-Seddon emphasized lower extremity
1967-Whiteside stressed 4 compartment fasciotomy
4. Demographics
Incidence:
Men 7.3/100,000
Women 0.7/100,000
69% due to trauma
36% fx tibia
9.8% distal radius
23% soft tissue injury without fx
10% on anticoagulants
High energy = low energy incidence
6. Casting & Wraps
Casting increases pressure 3-7 times
Positioning may effect pressure
Leg best position 0-37° plantar flexion
Elevation of extremity changes A-V gradient
7. Traction
Pressure increases linear with increasing weight
Posterior compartment of leg most effected
1 kg added weight
5% increase in posterior compartment
<2% increase in anterior compartment
Calcaneal traction increases dorsiflexion
11. Variables to Consider
Vascular tone
Blood pressure
Duration of elevated pressure
Metabolic demand of tissue
Lowered ischemic threshold of damaged muscle
12. Myoglobinemia
Released in high levels at reperfusion
Toxic to glomeruli
Metabolic acidosis & hperkalemia
Together lead to:
Renal failure
Cardiac arrhythmia & failure
Hypothermia
Shock
14. Clinical Diagnosis
The six ‘Ps’:
Pressure
Pain
Paresthesia
Paralysis
Pallor
Pulselessness
15. Pressure
Early finding
Only objective finding
Refers to palpation of compartment and its
tension or firmness
16. Pain
Classically out of portion to injury
Exaggerated with passive stretch of the involved
muscles in compartment
Earliest symptom but inconsistent
Not available in obtunded patient
17. Paresthesia
Also early sign
Peripheral nerve tissue is more sensitive than muscle
to ischemia
Permanent damage may occur in 75 minutes
Difficult to interpret
Will progress to anesthesia if pressure not
relieved
18. Paralysis
Very late finding
Irreversible nerve and muscle damage present
Paresis may be present early
Difficult to evaluate because of pain
19. Pallor & Pulselessness
Rarely present
Indicates direct damage to vessels rather than
compartment syndrome
Vascular injury may be more of contributing
factor to syndrome rather than result
21. Whiteside Technique
Simple technique
Readily available supplies
With 18 gauge needle least accurate
More accurate if use side port needle
22. Slit Catheter
Developed by Rorabeck
Considered ‘gold standard’
Need the catheter
Can use the measuring unit for Stic system
Can leave indwelling for continuous monitoring
23. Stryker Stic System
Easy to use
Can check multiple compartments
Different areas in one compartment
25. What is Critical Pressure?
>30 mm Hg as absolute number (Roraback)
>45 mm Hg as absolute number (Matsen)
<30 mm Hg for ∆p (where ∆p =diastolic
pressure – compartment pressure, McQueen)
<40 mm Hg for ∆P (where ∆P mean arterial
pressure* – compartment pressure, Heppenstall)
*mean arterial pressure is diastolic pressure plus 1/3 of
pulse pressure
27. Treatment
Lower leg to level of the heart
Remove cast
Split all dressings down to skin
Fasciotomy if continued clinical findings and/or
elevated compartment pressure